Researchers discovered that through temporary increases in brain malleability and simultaneous desensitization to fear memories, they could control fear responses in mice. This was achieved by inhibiting the Acan gene, thereby decreasing the presence of the protein Aggrecan, which solidifies and reduces malleability in the brain. The findings point towards potential new avenues for the treatment of post-traumatic stress disorder (PTSD).
Researchers discovered that through temporary increases in brain malleability and simultaneous desensitization to fear memories, they could control fear responses in mice. This was achieved by inhibiting the Acan gene, thereby decreasing the presence of the protein Aggrecan, which solidifies and reduces malleability in the brain. The findings point towards potential new avenues for the treatment of post-traumatic stress disorder (PTSD).
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